I am seeing more studies questioning the classification of Covid19 primarily as a respiratory illness.
A large number of patients are dying suddenly from heart attacks, hypoxia, aneurysms or blood clots after remission and recovery from respiratory ailments and after clear RT-PCR tests based on nasal swabs. Often
silent hypoxia it should be noted, without difficulty breathing.
As such it could also be classified as a cardio-vascular disease as well as a respiratory one.
This is discussed in depth here:
"Dr. Seheult explains the theory that seems most plausible to us here at MedCram.com: That COVID-19 is primarily a disease of the endothelium (the single layer of cells that line blood and lymphatic vessels). This hypothesis helps explain the increase in cardiovascular complications from COVID-19: strokes, myocardial infarctions, thromboembolism (pulmonary embolism, DVTs, and other blood clots), as well as many critical patients who do not appear to have classic ARDS symptoms (and have relatively normal lung compliance etc. as observed by the research of Dr. Gattinoni in Italy and others). A deep understanding of this theory requires a review of some foundational biochemistry including the electron transport chain and ATP - the energy currency of the body."
And reports such as ... (yes only reports and only DM but see quotes....)
"New York City Emergency Medical Services (EMS) Chief Lillian Bonsignore told
ABC News: 'It was astounding. We went from normal to wartime EMS in a week. And then it was just explosive and it continued."
"'We went from about 70 or 80 [cardiac arrests] a day which is what we normally do, to almost five times the amount,' Bonsignore said. 'I really was very surprised. I've never experienced a global pandemic.'
New York City EMS call data broken down by zip code shows that the increase in cardiac arrest calls and deaths aligned with the increase in COVID-19 cases.
Dr Fred Jacobs, the former commissioner of health in New Jersey, told ABC News that the 'data is showing an increase in cardiac arrest calls in the areas that also show an increase in COVID-19. So the question will be, "Is it causative?".'"
"But COVID-19 also has a unique weapon that allows it to attack the heart.
'With COVID specifically, what you see that you don't with the flu, is because under a microscope, coronavirus has all these spikes coming out of it, and those spikes are little proteins that are looking for receptors on the cells that they attach onto,' Dr Bonow explained.
'It's specifically looking for receptors in the lungs, but those same receptors sit on blood vessels, so it can attach on the lungs but also on blood vessels.'
Once they dock onto these blood vessel cells, the viral particles can trigger damage to these as well as to heart muscle, Dr Bonow says.
They can trigger 'hypercolagual states,' causing blood clots that lead to heart attacks.
In younger and healthier coronavirus patients, the heart muscle itself may become infected, a condition called mycarditis. "
New York City's heart attack spike could have been early sign of COVID-19 | Daily Mail Online
This study discusses the high incidence of clots with Covid 19 sufferers.
https://www.thrombosisresearch.com/article/S0049-3848(20)30120-1/pdf
This is not conclusive (of course) but it is looking more and more to me as if the effects outside of the lungs are significant enough for the classification of SARS Cov 2 to be broadened.
Also saw this just now
ts = 24m45s